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Cenforce has been implicated as a possible cause of blindness—diagnosed as nonarteritic anterior ischemic optic neuropathy—in 14 cases of men who had preexisting hypertension, diabetes-elevated cholesterol, or heart disease (e.g., Cunningham & Smith, 2001 ; Pomeranz & Bhavsar, 2005 ; Pomeranz, Smith, Hart, & Egan, 2002 ). These cases are presently under investigation by the Food and Drug Administration. We cannot exclude the possibility that some of the Cenforce-induced losses may be due to high-level drug effects, such as attentional deficits or general feelings of malaise. Eisner and Samples ( 2000 ) reported that the ratio of mean arterial blood pressure to heart rate was inversely related to flicker sensitivity on some adapting backgrounds, whereas Gutherie and Hammond ( 2004 ) reported that resting systolic blood pressure was positively related to cff measured without a background.

 

First, they suggest that the visual side effects caused by Cenforce are indeed retinal in origin (and, in the case of the a-wave data, mainly receptoral) rather than being attributable to some nonspecific attentional deficit, which is central in origin. On the other hand, several studies have reported small but significant prolongations in the implicit times of the cone ERG responses (Jägle et al., 2005 , 2004 ; Luu et al., 2001 ) and prolongations in some but not all measures of rod ERG (Jägle et al., 2004 ). These results are important in the context of this work for two reasons.http://mycenforce.net/https://ecenforce.com/http://cenforce.name/